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Baker-Gordon Syndrome

OMIM ID:

autosomal dominant

Baker-Gordon Syndrome

Alternate Names

BAGOS

Defective Genes

SYT1

Clinical Characteristics

Ocular Features

Poor visual acuity described as central in origin with poor eye contact.  Periorbital anomalies of low-set eyebrows and epicanthal folds are common.  The eyes have been described as “almond-shaped”.  Strabismus and nystagmus are commonly present.

Systemic Features

The facial features ae described as “fine” with a short nose and a thin upper lip.  The forehead is unusually high. 

There is general developmental delay with impaired intellectual development, delayed or absent walking, and behavioral psychiatric manifestations such as stereotypic and unpredictable outbursts.   There are often involuntary and hyperkinetic movements with dystonia, dyskinesia, ataxia and choreoathetosis.  The EEG is often abnormal although seizures have not been reported.

Genetics

Inheritance

De novo heterozygous mutations in the SYT1 gene (12q21.2) have been associated with this condition.  

Pedigree

Autosomal dominant

Autosomal dominant disorders require only one mutation for the disease to be expressed.  Since an affected parent has two chromosomes, only one of which has the mutant gene, parents can expect that half (50%) of their children will receive that one and inherit the disease.  It is common for individuals that inherit the mutation, however, to not have evidence of the disease (nonpenetrance).

Image
Sample pedigree of autosomal dominant inheritance

Autosomal dominant inheritance leads to a vertical pattern of transmission

Treatment & Management

No treatment has been reported.

Selected Resources

Web Resources

Publications

Displaying 1 - 2 of 2

Identification of a human synaptotagmin-1 mutation that perturbs synaptic vesicle cycling

PubMedID: 25705886

SYT1-associated neurodevelopmental disorder: a case series

PubMedID: 30107533