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GM3 Synthase Deficiency

OMIM ID:

autosomal recessive

GM3 Synthase Deficiency

Alternate Names

Amish infantile epilepsy syndrome
SPDRS
salt and pepper mental retardation syndrome

Defective Genes

SIAT9

Clinical Characteristics

Ocular Features

Optic atrophy is the primary ocular feature in this disorder.  ERG amplitudes and retinal pigmentation are normal.  Visual impairment is pronounced with no reactions to threatening visual stimuli.  Eye movements are random and uncoordinated.  Optic atrophy is present but no retinal abnormalities have been reported.

Systemic Features

Infants may appear normal at birth but within a few months develop signs of developmental stagnation with onset of tonic-clonic seizures.  Irritability, poor feeding, vomiting and failure to thrive are important features.  Generalized hypotonia is evident but lower limb deep tendon reflexes may be present.  Normal developmental milestones are never achieved and patients are unresponsive to their environment.  Older individuals develop non-purposeful choreothetoid movements.  The EEG shows multifocal epileptiform discharges and brain MRIs show diffuse atrophy in older patients.

Hypo- and hyperpigmented skin macules in a 'salt and pepper' pattern, have been described.  These vary from 2-5 mm in size and are located primarily on the extremities.  These are found among children older than 3 years of age and some parents have reported that the hyperpigmentation may decrease after many years.  No such lesions were found in mucosal tissue.        

Genetics

Inheritance

This is an autosomal recessive disorder secondary to homozygous mutations in (ST3GAL5) (2p11.2) encoding sialytransferase (SIAT9).

The nonsense mutation results in a deficiency of functional GM3 synthase important in the utilization of lactosylceramide necessary for the production of downstream gangliosides.

Pedigree

Autosomal recessive

In order for autosomal recessive disorders to be expressed, offspring generally must inherit two mutations, one from each carrier parent.  Carriers with only one mutation, such as the parents, do not have clinical disease.  Note that carrier parents can expect that 1 in 4 children (25%) will inherit both mutations and have the disorder, 2 in 4 children (50%) will be carriers like their parents, while 1 in 4 children (25%) inherit neither mutation.

Image
Sample pedigree of autosomal recessive inheritance

In order for autosomal recessive disorders to be expressed, offspring generally must inherit two mutations, one from each carrier parent. Carriers with only one mutation, such as the parents, do not have clinical disease. Note that carrier parents can expect that 1 in 4 children (25%) will inherit both mutations and have the disorder, 2 in 4 children (50%) will be carriers like their parents, while 1 in 4 children (25%) inherit neither mutation.

Treatment & Management

There is no known treatment for the enzyme deficiency.  Seizures respond poorly to anti-epileptic medications.

Selected Resources

Web Resources

Publications

Displaying 1 - 3 of 3

Etiology of Vision Loss in Ganglioside GM3 Synthase Deficiency

PubMedID: 17050284

GM3 synthase deficiency due to ST3GAL5 variants in two Korean female siblings: Masquerading as Rett syndrome‐like phenotype

PubMedID: 27232954

Infantile-onset symptomatic epilepsy syndrome caused by a homozygous loss-of-function mutation of GM3 synthase

PubMedID: 15502825