Skip to main content

Ataxia and Polyneuropathy, Adult-Onset

OMIM ID:

mitochondrial

Ataxia and Polyneuropathy, Adult-Onset

Defective Genes

MT-ATP6

Clinical Characteristics

Ocular Features

This condition has its onset in young adults.  Early ocular signs are gaze-evoked horizontal nystagmus and defective ocular pursuit movements with the full range of extraocular movements.  Some patients but not all have optic atrophy.  Ptosis is not present.

Systemic Features

Gait disturbances have their onset in the first or second decades of life.  The gait may be broad-based.  Intermittent hemiparesis with headache, nausea and vomiting has been reported in some individuals.  Absent ankle jerks and extensor plantar responses have been noted but general muscle tone and strength is usually normal.   An axonal sensorimotor neuropathy may be present in midlife as documented by nerve conduction studies.  MRIs of the brain may reveal cerebellar atrophy.

Mild cognitive problems have been reported in a few individuals.

Genetics

Inheritance

This is a mitochondrial disorder secondary to mutations in the mitochondrial MT-ATP6 gene.

Treatment & Management

No treatment has been reported.

Selected Resources

Web Resources

Publications

Displaying 1 - 2 of 2

Adult‐onset ataxia and polyneuropathy caused by mitochondrial 8993T→C mutation

PubMedID: 16049925

Episodic ataxia and hemiplegia caused by the 8993T→C mitochondrial DNA mutation: Figure 1

PubMedID: 18055910